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Juris Bunkis and Dr
Historically described as mere fat-solubilizing agents, these amphipathic compounds were recently recognized as key signaling molecules capable to modulate the host metabolism directly acting as ligands of intestinal GPCRs (101, 338, 339), or after being metabolized by the colonic microbiota into secondary bile acids, mostly deoxycholic and lithocholic acid(340)
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FAK-paxillin signaling primarily explains adhesion turnover, cytoskeletal reorganization, and directional movement
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