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Association of GLP-1 receptor agonists with chronic obstructive pulmonary disease exacerbations among patients with type 2 diabetes
They represent the immune system responding to the physical trauma of needle penetration, the chemical properties of the medication, or both
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Fin56-induced ferroptosis is supported by autophagy-mediated GPX4 degradation and functions synergistically with mTOR inhibition to kill bladder cancer cells
Retrieved from Li, Z., Wang, Y., Xu, Q., et al
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