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Archivado desde el original el 28 de abril de 2007
( 11 ) provide compelling evidence that GLP-1 signaling in the CNS also serves to increase noninsulin-dependent glucose uptake by muscle and stimulates insulin secretion via neural connections to these tissues.
Some studies have shown poorer outcomes for breast, colon, and pancreatic cancers when TLR4 is overexpressed (63, 64)
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