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This process can be triggered by intrinsic pathways, including caspase activation and mitochondrial cytochrome C release, as well as by extrinsic pathways, such as TNF receptor engagement ( Another critical mechanism in EBI is autophagy, a process essential for neuronal survival that recycles damaged organelles and cellular debris following ischemic injury ( Pyroptosis is a form of inflammatory cell death that is critically dependent on the activation of the NLRP3 inflammasome

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