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Complete prevention requires maintaining GLP-1 medication long-term, similar to how blood pressure control requires ongoing antihypertensive therapy
They tend to modulate rather than completely block pathways
Decrease of Cav-1 binding released the eNOS and subsequently enhanced the activation of eNOS
I would also like to thank David L Ludwig for valuable discussions about the physiology and pharmacology of GLP-1 and for careful reading and commenting on the manuscript
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