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Impaired mitochondrial function in dopaminergic neurons in the substantia nigra can lead to increased oxidative stress, neuronal damage, and neuronal death, contributing to PD progression [20]
fMRI language dominance and FDG-PET hypometabolism
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We recently reported that ALCAR was able to act on endothelial cells interfering with the VEGF/VEGFR2 and CXCR4/CXCL12 axes and reducing angiogenesis in vitro
Its seen as a crutch or the easy way out, when that couldnt be further from the truth
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