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As a triple incretin receptor agonist, it: Activates GLP-1 receptors , reducing appetite, slowing gastric emptying, and improving insulin secretion Activates GIP receptors , enhancing insulin response and potentially supporting fat metabolism Activates glucagon receptors , increasing energy expenditure and hepatic glucose output Whilst glucagon receptor activation increases hepatic glucose output, the net glycaemic effect of retatrutide is mitigated by the concurrent GLP-1 and GIP activity, resulting in overall improvements in glycaemic control in trial participants
10.1111/j.1399-3054.1989.tb05668.x 158 TiwariS.LataC.ChauhanP
The concern isn't unfounded: GLP-1 receptor agonists create sustained metabolic shifts that increase oxidative byproducts during fat oxidation, and glutathione is the body's primary intracellular antioxidant
The brains of AD patients invariably show cholinergic deficits, characterized by decreased levels of acetylcholine, choline acetyltransferase, and/or nicotinic acetylcholine receptors (Mayeux and Stern 2012)
Safety and Considerations When considering treatments like NAD+ and glutathione, particularly for conditions as complex and varied as long COVID, understanding their safety profile and any considerations is crucial
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