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[3] Additional in-vitro studies suggest that GHK-Cu may down-regulate transforming growth factor-1, a cytokine implicated in premature follicle regression, thereby potentially prolonging the anagen (growth) phase

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[6] Because systemic absorption of topically applied GHK-Cu seems minimal, clinically significant pharmacokinetic interactions with oral medications are unlikely

By inhibiting this enzyme, 5-Amino-1MQ prevents the loss of these precursors, effectively redirecting them into the NAD+ salvage pathway

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