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It has a docking effect with Src homology 2 signal molecules in cells, transferring the signal through Src homology 3 to Son of Sevenless, phosphorylating downstream Ras molecules, thereby activating the extracellular signal-related kinases (ERK1/2) pathway and regulating the growth, proliferation, and differentiation of osteoblasts [26, 27]

More pathways means more variables

The pathology culminates in an immune-mediated rearrangement and destruction of the synovium in joints, causing a range of painful mobility-impairing symptoms

LWT 142

The Role of Chondrocyte Hypertrophy and Senescence in Osteoarthritis Initiation and Progression