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Mitochondria and mitochondrial DNA: key elements in the pathogenesis and exacerbation of the inflammatory state caused by COVID-19

As research progresses, regulated pharmaceutical development may eventually offer validated pathways for NNMT inhibitors

The pathophysiology of Long COVID, myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS), and related dysautonomias shares a striking commonality: severe, persistent post-viral metabolic dysfunction

A multimodal approach is likely needed one that combines medication with nutrition support, physical activity, and ongoing clinical oversight