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Among various indicators, glutathione reductase (GR), a key enzyme in the oxidative stress process, shows activity changes that can signal hepatocyte metabolic stress and functional abnormalities earlier, providing an important diagnostic window for identifying early liver damage [3]
(2025): Clonidine
Further supporting this, Lock and colleagues demonstrated that glutathione protects against 2-chloropropionic acid (CPA)-induced cerebellar toxicity by preserving thiol redox equilibrium and potentially interacting with NMDA receptor-mediated pathways
doi: 10.1016/S0140-6736(20)32511-3 Summary Keywords chronic liver disorder, metabolic syndrome, non-alcoholic fatty liver disease, oxidative stress, oral glutathione Citation Santacroce G, Gentile A, Soriano S, Novelli A, Lenti MV and Di Sabatino A (2023) Glutathione: Pharmacological aspects and implications for clinical use in non-alcoholic fatty liver disease
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