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10.1371/journal.pgen.1000361 PLoS Genet
T2DM is a common and complex but highly treatable disease associated with a multitude of devastating complications such as retinopathy, neuropathy, coronary artery disease, cerebrovascular disease, peripheral vascular disease, and nephropathy [1]
Thus, the clinical and experimental studies convincingly show the involvement of mitochondrial abnormalities in RS, although the mechanisms underlying the role of mitochondrial dysfunction need further investigation
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