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Oral N -acetyl-cysteine attenuates loss of dopaminergic terminals in -synuclein overexpressing mice
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Its effects contribute to improved tissue adaptability and long-term structural integrity
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Considering the link between vascular dysfunction and overt CVD, as well as the role of cellular senescence in mediating vascular aging, it is highly plausible that cellular senescence-induced vascular aging is responsible for the early CV-related morbidity and mortality observed in these patients
