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10.3390/antiox5030030 55 LiuB.MaoQ.CaoM.XieL
M.SchuitR
The advantage is that those risks are more predictable and more manageable than the systemic disruption caused by broad-acting compounds
Studies have shown that in the HF mouse of myocardial infarction models, the depletion of neutrophils through antibody-mediated or genetic methods can halt the progression of HF, especially the left ventricular remodeling and fibrosis progression in the middle and late stages of HF (135), further indicating that neutrophils are a potential therapeutic target for lowering myocardial damage and preventing HF (159, 160)
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