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Studies suggest they may influence telomere dynamics, oxidative stress markers, and turnover pathways, with research pointing to potential roles in maintaining signaling balance during aging
Vitamin B12 Deficiency: 12 , 12
Consistent with the view that remodeling is context dependent, therapy-induced senescence has been reported to increase MERCS' contact surface while reducing ER-mitochondria Ca2+ flux because of decreased IP3R expression and reduced IP3R1-VDAC1 interaction (235)
In vivo mitochondrial function in idiopathic and genetic parkinson's disease
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