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DR retinal chronic inflammation amplifies RPE ferroptosis: inflammatory factors activate NOX to produce ROS via NADPH, damaging antioxidant systems and inducing ferritinophagy to release free Fe + , further enhancing ferroptosis signals (150)

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This research demonstrates BPC-157s effects on tendon healing through enhanced fibroblast migration and survival

This clinical challenge has stimulated a search for additional mechanisms outside canonical cytokine signaling