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As dietary fatty acids are assembled into lipoproteins in intestinal enterocytes for subsequent circulatory transport, blocking lipoprotein transit from endoplasmic reticulum (ER) to Golgi by Pluronic L-81 in rats robustly reduces (4.5-fold) lymph GIP levels and secretion rates in response to Liposyn to levels similar to saline controls (Lu et al., 2012)
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