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These results indicate that the process of disulphidebond formation might occur independently of GSSG

Under basal conditions, NRF2 is kept inactive through binding to its negative regulator KEAP1, which is a redox-regulated substrate adaptor for the Cullin (Cul)3-RING-box protein (Rbx)1 ubiquitin ligase complex that directs NRF2 for degradation [10]

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doi:10.1525/fq.2018.72.2.67

Similarly, bioinformatic models using eight key ferroptosis-related gene signatures, or a six-biomarker diagnostic signature centered on TP53, have demonstrated exceptionally high diagnostic accuracy in distinguishing DKD from controls (231, 232)