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This indicates the existence of a GGCT-mediated Gln metabolic shunt in tumor cells: Gln is prioritized for conversion to GSH, which scavenges ROS, alleviates oxidative stress, and ultimately promotes tumor cell proliferation
Another autophagy inhibitor 3-MA also produced rescuing effects but to a smaller extent in erastin-treated cells
However, with continued exposure, immune activation may fail to resolve appropriately
10.1016/j.addr.2021.113851 64
Peptide-spectrum match (PSM) validation was carried out using the percolator algorithm to ensure reliable identifications
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