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Discussion In the present study, we demonstrated that permanent IGF-1 attenuated oxidative SIPS of hepatocytes via inhibiting the interaction between nuclear p53 with progerin (Fig
By reducing IGFBP-3 binding, more of the administered peptide reaches IGF-1 receptors as free, biologically active ligand
Similar content being viewed by others Introduction G protein-coupled receptors (GPCRs) and their immediate transducersheterotrimeric G proteinshave been the subject of intensive scrutiny for decades, primarily due to their pivotal roles in innumerable physiological and pathological processes 1
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