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Pharmacological blockade of CB1R prevents insulitis progression in humans In light of a dysregulation in the ECS occurring in type 1 diabetes, with increased expression of CB1R in immune cells and beta cells, we investigated its role in insulitis

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demonstrated that TMAO activates NF-B in aortic endothelial cells via mitogen-activated protein kinase (MAPK)/extracellular regulated protein kinases (ERK) signaling, promoting leukocyte adhesion and vascular inflammation (82)

Gruener, S., et al

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