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It appears that the increase in HO-1 following SYN-treatment is in response to oxidative stress emanating from glial activation by misfolded -synuclein but following SYN DA or Buffer DA increased HO-1 is a glial response to oxidized dopamine
It does not stimulate muscle protein synthesis on its own, and it does not act on androgen receptors
As a result, glucose, which requires less oxygen than FAs to produce ATP, becomes the primary energy source for the heart [30]
That is why Ishin C Plus and Ishin Premium were designed to support balance not overload
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