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Polymorphisms in SLC37A3, SLC38A8, and SLC39A8 have been implicated in NAFLD progression, but their specific regulatory mechanisms in fibrosis progression have yet to be fully clarified

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It also modulates the activity of metalloproteinases (enzymes that break down extracellular matrix proteins) and their inhibitors, creating a balanced environment where damaged tissue is cleared while new tissue is being built

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This allows for versatility in addressing different areas of the face and achieving various outcomes.