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We used an inducible model that with administration of tamoxifen allowed us to temporally delete the IGF1R after Nestin + cells in the brain had reached the quiescent phase but at a time point in which the skeleton continues to undergo rapid growth
These effects were generally mild and transient
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Canonical and non-canonical WNT signaling in cancer stem cells and their niches: Cellular heterogeneity, omics reprogramming, targeted therapy and tumor plasticity (Review)
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