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4.4 Neonatal hypoxicischemic encephalopathy The pathophysiology of HIE involves hypoxicischemic injury, characterized by perturbations in mitochondrial dynamics and the inhibition of critical energy-producing pathways, including oxidative phosphorylation and fatty acid -oxidation (54)

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77 Pharmacological inhibition of PDEs could theoretically sustain or elevate cAMP levels, potentially enhancing GLP-1 RA signaling

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