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Low v-ATPase-inhibition increases m and enhances cell fitness under mitochondrial mistranslation in multiple cell-types and mitochondrial disease patient-derived fibroblasts To confirm that the suppressed cell growth defect under ACT-induced mistranslation was due to v-ATPase deficiency, we used the highly selective v-ATPase inhibitors concA and bafA on WT cells
Erythromycin, 3-azido-3-deoxythymidine, 5-iodo-2-deoxyuridine, (+)-catechin hydrate, l-adrenaline, desloratadine, zoledronic acid monohydrate, testosterone, estriol, and oxaliplatin were purchased from Tokyo Chemical Industry Co., Ltd and used as received
Pan L, Lu MP, Wang JH, Xu M, Yang SR (2020) Immunological pathogenesis and treatment of systemic lupus erythematosus
As a refresher, your body naturally makes a GLP-1 hormone that triggers the release of insulin
Socha E, Koliski P, Koba M, et al
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