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Since mitochondrial function is frequently defective in populations of human spermatozoa [56], it is possible that this trans-plasma membrane redox system is up-regulated in defective human spermatozoa in a similar fashion to the enhanced activity recorded in Rho O cells in order to maintain the redox status of the NAD + /NADH couple
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Inflammatory Bowel Diseases , 14(3), 324331
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