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16 Berenbaum hypothesized that the therapeutic potential of GLP-1 in OA may arise from targeting the crosstalk between macrophages and fibroblasts, which are known to drive inflammation, fibrosis, and downstream cartilage damage
And that's how we made drugs for centuries
Notes: Monitor subjects glucose levels to prevent hypoglycemia
B.StegmannB
They provide a clear link between biochemical disruptions, such as dopamine depletion and neuroinflammation, and the resulting cognitive, psychiatric, and motor symptoms observed in chronic METH abusers
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