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Work in the quinolinic acid lesion model of Huntingtons disease reported that the IGF-1 amino-terminal tripeptide GPE is neuroprotective to striatum, and a body of medicinal chemistry followed, generating analogues designed for better stability and bioavailability
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(iv) Reversing retinal vascular leakage as seen in fluorescein angiograms
I., Dobrinskikh, E., Limesand, S
Tripartite motif 32 prevents pathological cardiac hypertrophy
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