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Some studies have identified metabolic deficiencies in autistic children [14], and it has recently been shown that there is a characteristic metabolic profile in many autistic children involving disturbances in methionine and glutathione metabolism [14, 15]

Presented for research literature review only Hsu CH et al

FIN56 is one ferroptosis inducer derived from CIL56, which is capable to induce cell death without activating caspases 3/7 (Shimada et al., 2016)

Lab markers for glutathione: Free Glutathione (GSH), Glutathione disulphide (GSSG) and GSH/GSSG ratio