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AMPK also phosphorylates STAT6 at Ser564 to increase STAT6 activation and arginase 1 production, suggesting that cholesterol 25-hydroxylase acts as an immune metabolic checkpoint that can manipulate macrophage fate to reshape CD8 + T-cell surveillance and antitumor responses
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In regard to TC, it is also pertinent to remind the involvement of chemokines (8998) and the peroxisome proliferator-activated receptors (PPARs) in the molecular oncogenesis (95106), so that both types of molecules may serve as novel targets of TC precision therapy or prevention
W., Guo, B
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