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Two estimands were employed to assess treatment efficacy, accounting for intercurrent events and missing data differently
The mechanism of action of DSePA against RP involved its ability to suppress the radiation-induced sustained inflammatory response by upregulating the pulmonary activity level of GPx and concurrently blocking the NF-kB/IL-17/G-CSF/neutrophil axis (Table 2) (Fig
GHK-Cu has no FDA-approved indication
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