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Importantly, the inhibition of BTK and SYK pathways, implicated in the amplification NF-B and MAPK-mediated inflammatory cascades and intersecting with the JAK/STAT network [246, 250], could support a translational link between B-cell-driven inflammation and attenuation of fibrosis in HS lesions

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Nevertheless, PS translocation to the outer leaflet of the phospholipid bilayer, a hallmark of eryptosis, can frequently occur without caspase-3 recruitment, primarily in response to Ca 2+ overload [84]

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