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Ballatori, N., Krance, S
Thus, FBP is ideally positioned to coordinate the regulation of cell adhesion with the metabolic control exerted by AMPK and to reinforce Rac1-dependent cell protrusion by keeping AMPK, a negative regulator of integrin activity and cell spreading, inactive
As long as LKB1-AMPK signaling is activated, the regulation of the metabolic branch of mTOR signaling cannot be impaired in spite of the abnormal of PI3K/AKT or receptor tyrosine kinase signaling (237)
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Implications of NAD+ boosters in translational medicine - Kang - 2020 - European Journal of Clinical Investigation - Wiley Online Library n.d
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