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Cytotoxic factors such as inflammatory mediators and nitric oxide are released from the M1 phenotype, which also disrupts the bloodbrain barrier (BBB), increases its permeability, and engulfs vascular endothelial cells, causing a breakdown of this barrier after stroke (Xu et al
Another target is PAD4 (peptidyl arginine deiminase 4), the enzyme required for NET formation
The dawn of a new healing therapy has arrived
10.1210/clinem/dgz324 50 SmitD
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