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CrossRef Deciphering the circadian rhythm in colorectal cancer: a bibliometric analysis of research landscape and trends Linzi Chen, Zhongjie Wang, Ningkun Xiao, Jinhui Liu, Yuhan Tao, Sifang Zhang Frontiers in Oncology .2025;[Epub] CrossRef Mechanism of activation of the Nrf2-Keap1 pathway by fermented Chinese herbal medicines via the gut-liver axis to alleviate cadmium-induced hepatotoxicity Li Jiang, Qiuhong Wu, Yilei Liang, Zhiwen Yang, Guang Fan, Pan Zhou, Xinyue Liu, Yachao Wang Environmental Chemistry and Ecotoxicology .2025

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From Gut Hormone to Wonder Drug If you strip away the hype, semaglutide is simply a clever copy of a natural hormone that tells the body youve eaten enough.[1] In the 1980s and 1990s, researchers discovered that the gut hormone GLP1 could lower blood sugar, slow digestion and curb appetite, but the natural hormone disappeared from the body within minutes.[2][3] So Novo Nordisk chemists spent years armouring GLP1tweaking its structure so it survived longer in the bloodstream and could be given as a weekly injection instead of a constant drip.[4][5] One of those tweaked versions was semaglutide, designed to stay active for around a week, which made it practical for realworld use in diabetes and, later, obesity.[6][7] Key point: A basic hormone signal from the gut was turned, stepbystep, into a longacting drug molecule that could be injected once a week

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