Growth factor upregulation
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Several complex signal transduction pathways and cellular processes have been implicated in APAP hepatotoxicity, including those involving c-Jun N-terminal kinase, Nuclear factor E2-related factor 2 (Nrf2), p53, endoplasmic reticulum stress, autophagy, and sterile inflammation (Chowdhury et al
References Autoimmune Diseases. National Institute of Environmental Health Sciences , www.niehs.nih.gov/health/topics/conditions/autoimmune
albicans , suggesting that when the two microorganisms co-infect the peritoneal cavity, there may be other processes independent of morphology that are regulated by Efg1, which result in fatal synergies [117, 118]