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Journal of Biomaterials Science
606 By activating autophagy and inhibiting ferroptosis, rapamycin reduces the loss of dopamine neurons in the substantia nigra and improves behavioral symptoms in PD model mice, suggesting that the mutual regulation of ferroptosis and autophagy may provide therapeutic targets for drug therapy of PD
Lung myofibroblasts promote macrophage profibrotic activity through Lactate-induced histone lactylation
Abstract Toxicity is not only a function of damage mechanisms, but is also determined by cellular resilience factors
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