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A molecular model of human branched-chain amino acid metabolism

The normalization of oxidative stress markers (MDA, SOD, and GSH) and inflammatory markers (IL-17, TNF-, IL-6, and CRP) by PTX-CSNPs suggested a synergistic mechanism: chitosans intrinsic antioxidant properties combined with PTXs ability to inhibit TNF- and oxidative pathways

These data suggest that compared to HFCS feeding alone, HFCS with inulin feeding increased hepatic fructose carbon use to generate mitochondrial acylcarnitine for FAO