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While sedated under ether anesthesia, deep partial skin thickness burns were induced on the back of each test subject through the method of control burning
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Interestingly, however, -arrestin1 promotes insulin secretion via direct interaction with EPAC2, thus favoring EPAC2-induced Rap1 activation and potentiating sulfonylurea-induced insulin release [43]
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However, gastrointestinal (GI) adverse events remain the most common limitations to long-term therapy
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