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Nutr Cancer 1995;17:57-75
Furthermore, we provide first data that the GSH level in human neurons may be mediated by NMDAR activation since the protective effect of enhanced network activity could be blocked with an NMDAR antagonist APV, consistent with the previous report (Fig
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Notably, mitochondrial dysfunction is an underlying component of ASC physiology that is sensitive to GC signaling and plays a central role in regulating HPA axis and innate immune responses
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