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Collectively, these results suggest a model in which TRIP12 regulates the steady-state level of OTUD5 protein, whose accumulation under TRIP12 depletion represses TNF-induced NF-B signaling partly through TRAF2-deubiquitylation (Fig
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doi: 10.1053/j.gastro.2015.09.004 49 MitaMSatohMShimadaAOkajimaMAzumaSSuzukiJSet al
Heme-containing dioxygenases
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