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Although mechanisms describing the interaction between the key pathway constituents Nrf2, Keap1 and p62 are widely reviewed and discussed in literature, quantitative dynamic models bringing together these mechanisms with time-resolved data are limited

Still, the findings offer clarity for patients and clinicians navigating a rapidly changing area of care

Aims and hypotheses Primary aim The primary aim is to test the hypothesis that treatment with 400 mg per day of oral SAMe for 180 days will lead to a greater reduction from baseline plasma levels of p-tau181 compared to placebo in patients with mild cognitive impairment or dementia due to AD

introducing how obesity and insulin resistance (initiating factors for the onset of MetS and diabetes) may be opposed by recombinant-human-IGF-1 (rhIGF-1) treatment